Sirolimus

Evidence Level: L2 Predicted Indications: 10

Table of Contents

  1. Sirolimus
  2. Sirolimus: From Organ Transplant Rejection Prophylaxis to Liposarcoma
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. India Market Information
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## Pharmacist Assessment Report

Sirolimus: From Organ Transplant Rejection Prophylaxis to Liposarcoma

One-Sentence Summary

Sirolimus (DrugBank DB00877) is an mTOR inhibitor globally established for prophylaxis of organ rejection after renal transplantation, though it is currently not marketed in India. The TxGNN model’s top-ranked candidate indication is Liposarcoma, supported by 5 clinical trials and 12 publications, but only one trial directly tests sirolimus itself in this disease — most other evidence comes from related mTOR inhibitors (temsirolimus, everolimus, ridaforolimus).


Quick Overview

Item Content
Original Indication Not documented in India regulatory data (drug not marketed); the molecule is globally established for prophylaxis of renal transplant rejection
Predicted New Indication Liposarcoma
TxGNN Prediction Score 99.89%
Evidence Level L2
India Market Status ✗ Not Marketed
Number of Registrations 0
Recommended Decision Hold

Why is This Prediction Reasonable?

A formal DrugBank-sourced mechanism-of-action record was not available for this evaluation (data gap DG002). However, the mechanistic role of sirolimus is well documented throughout the collected literature and trial descriptions: it inhibits mammalian target of rapamycin (mTOR), a serine/threonine kinase that integrates growth-factor and nutrient signaling to drive cell proliferation, growth and survival (e.g., PMID 37400145: “Rapamycin (RAPA) is an inhibitor of mTOR”).

The connection between sirolimus’s original use (immunosuppression in transplantation) and the proposed new indication (liposarcoma) runs through this same mTOR pathway. Dedifferentiated liposarcoma has been shown to exhibit constitutive activation of the Akt-mTOR and MAPK pathways (PMID 26518767), providing a biological rationale for mTOR blockade as an anti-proliferative strategy. This has motivated a class-wide interest in “rapalogs” (sirolimus, temsirolimus, everolimus, ridaforolimus) across sarcoma subtypes, illustrated by several trials in the evidence pack (NCT00093080, NCT03114527, NCT00949325, NCT01614795).

Only one trial directly evaluates sirolimus itself in this indication — NCT02821507, a Phase 2 study of sirolimus + cyclophosphamide in metastatic/unresectable myxoid liposarcoma and chondrosarcoma (n=70, Grade A relevance). The remaining trials and much of the literature test related rapalogs, so the current evidence base for liposarcoma is best characterized as class-effect support with limited drug-specific confirmation.

Portfolio note: Among the 10 candidate indications surfaced for sirolimus in this evidence pack, two reach a substantially stronger evidence tier (L1, decision stage S3, “Proceed with Guardrails”): lymphangiomyoma/LAM (rank 5) and lung PEComa (rank 9). Both are underpinned by completed Phase 3 RCTs of sirolimus itself (MILES trial, NCT00414648) and an existing FDA approval (2015) for LAM. Given that sirolimus has zero current registrations in India, these two indications likely represent a more mature and lower-risk repurposing opportunity than liposarcoma and warrant separate evaluation.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT02821507 Phase 2 Completed 70 Sirolimus + cyclophosphamide in metastatic/unresectable myxoid liposarcoma and chondrosarcoma — direct sirolimus evidence in this indication
NCT00093080 Phase 2 Completed 216 Ridaforolimus (AP23573), an mTOR inhibitor, in advanced sarcoma (QDx5 every 2 weeks regimen)
NCT01614795 Phase 2 Completed 46 Cixutumumab (IGF-1R antibody) + temsirolimus in pediatric recurrent/refractory sarcoma
NCT03114527 Phase 2 Active, not recruiting 48 Ribociclib + everolimus in advanced dedifferentiated liposarcoma and leiomyosarcoma
NCT00949325 Phase 1/2 Completed 24 Temsirolimus (Torisel) + liposomal doxorubicin in advanced soft tissue/bone sarcoma

Literature Evidence

PMID Year Type Journal Key Findings
37967116 2024 RCT (Phase 2) Clin Cancer Res SAR-096: Ribociclib + everolimus in advanced dedifferentiated liposarcoma and leiomyosarcoma
39796641 2024 Review Cancers Overview of novel therapeutics in soft tissue sarcoma, including mTOR-pathway agents
37222206 2023 Review Curr Opin Oncol Rationale and results of molecular-targeted agent trials in advanced sarcomas
20497911 2010 Review Bull Cancer Targeted treatment of rare connective tissue tumors and sarcomas, including mTOR-directed approaches
16434506 2006 Cohort J Am Soc Nephrol Sirolimus after early cyclosporine withdrawal reduces cancer risk in renal transplant recipients
26518767 2016 Mechanistic/Preclinical Tumour Biol Akt-mTOR and MAPK pathway activation demonstrated in dedifferentiated liposarcoma specimens
37400145 2023 Preclinical Cancer Genomics Proteomics Chloroquine + rapamycin combination effective against well-differentiated liposarcoma models
36309387 2022 Preclinical In Vivo Chloroquine + rapamycin arrests tumor growth in a dedifferentiated liposarcoma PDOX mouse model
25519700 2015 Preclinical Mol Cancer Ther MLN0128, an ATP-competitive mTOR kinase inhibitor, shows antitumor activity in bone/soft-tissue sarcoma
32711543 2020 Case report Diagn Pathol Fibro-adipose vascular anomaly (FAVA) cases with mTOR pathway activation; sirolimus response noted

India Market Information

Sirolimus currently has no registered products in India (market status: Not Marketed; total registrations: 0). No license or dosage-form data is available for this evaluation.


Safety Considerations

Drug Interactions: Sirolimus has 645 documented interactions on record. Notable Major-severity interactions include:

  • Amphotericin B / Amphotericin B (lipid complex)
  • Clarithromycin
  • Mesalazine
  • Balsalazide

Numerous Moderate-severity interactions are also documented, including with corticosteroids (hydrocortisone, dexamethasone, betamethasone, budesonide, triamcinolone), antidiabetic agents (metformin, pioglitazone, acarbose, alogliptin, canagliflozin, dapagliflozin, chlorpropamide, albiglutide), aprepitant, and cimetidine.

Drug-specific key warnings and contraindications (TFDA/local package insert equivalent) were not available for this evaluation (data gap DG001, flagged as Blocking — this prevents completion of the S1 safety pre-screen).


Conclusion and Next Steps

Decision: Hold

Rationale: Evidence for sirolimus in liposarcoma reaches only L2 (a single direct sirolimus trial, n=70; remaining support is class-effect from related rapalogs), and local safety documentation (warnings/contraindications) is currently a blocking data gap. This combination does not yet meet the bar for proceeding, even with guardrails.

To proceed, the following is needed:

  • TFDA-equivalent package insert warnings and contraindications (DG001 — blocking, required for S1 safety screen)
  • Formal DrugBank/verified mechanism-of-action documentation (DG002)
  • Additional direct sirolimus (not rapalog-class) trial data in liposarcoma, ideally Phase 2/3
  • Separately, given the absence of any India registration for sirolimus, consider prioritizing a parallel evaluation of lymphangiomyoma/LAM and lung PEComa (both L1 evidence, existing FDA-approved precedent) as potentially faster-track repurposing candidates for this molecule

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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