Cyclandelate

Evidence Level: L2 Predicted Indications: 10

Table of Contents

  1. Cyclandelate
  2. Cyclandelate: From Peripheral Vascular Disease to Migraine Prophylaxis
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. India Market Information
    7. Safety Considerations
    8. Other Predicted Indications (Screened, Lower Priority)
    9. Conclusion and Next Steps
    10. Disclaimer

## Pharmacist Assessment Report

Cyclandelate: From Peripheral Vascular Disease to Migraine Prophylaxis

One-Sentence Summary

Cyclandelate (brand name Cyclospasmol) is a peripheral vasodilator historically used for circulatory disorders such as peripheral vascular disease and intermittent claudication. Among 10 TxGNN-predicted indications, migraine disorder stands out as the best-supported candidate, with a TxGNN score of 99.69% and 6 randomized controlled trials (13 publications total) directly evaluating cyclandelate for migraine prophylaxis — making it the only candidate in this evidence pack that clears an L2 evidence bar.

(Note: this drug generated 10 TxGNN-predicted indications. This report focuses on the strongest-evidenced one, migraine disorder. A summary of the other 9 lower-priority candidates appears near the end.)


Quick Overview

Item Content
Original Indication Peripheral Vascular Disease / circulatory disorders (historical, brand name Cyclospasmol) — no structured Taiwan/India license record exists
Predicted New Indication Migraine disorder
TxGNN Prediction Score 99.69% (rank 5,863 of candidates)
Evidence Level L2
India Market Status ✗ Not marketed
Number of Registrations 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

Currently, no formal DrugBank/regulatory mechanism-of-action record is available for cyclandelate (flagged as a data gap in this evidence pack). Based on the pharmacological description found in the supporting literature (PMID 8902255), cyclandelate inhibits calcium-induced contraction of vascular smooth muscle, inhibits platelet aggregation induced by thrombin, platelet-activating factor and adenosine, and suppresses provoked serotonin (5-HT) release from platelets. This places it functionally alongside calcium-channel-blocking migraine prophylactics such as flunarizine.

Cyclandelate’s original use was as a peripheral/cerebral vasodilator for circulatory disorders (Cyclospasmol). Migraine pathophysiology involves both vascular (cortical spreading depression, vasospasm) and serotonergic mechanisms — both of which overlap with cyclandelate’s known pharmacology. This mechanistic bridge is why cyclandelate was actually studied and used as a migraine prophylactic agent in several European countries during the 1980s–1990s, predating this TxGNN prediction by decades — i.e., the model’s high score recovers a real, previously established clinical use rather than a purely novel hypothesis.

It’s worth noting the evidence is mixed: several earlier RCTs (1987–1998) reported positive prophylactic effects comparable to propranolol, pizotifen, and flunarizine, but the largest and most recent placebo-controlled trial (PMID 11298666, 2001, n=251) failed to meet its primary endpoint. This tempers the otherwise favorable historical signal.


Clinical Trial Evidence

Currently no related clinical trials registered on ClinicalTrials.gov or ICTRP.


Literature Evidence

PMID Year Type Journal Key Findings
1573338 1992 RCT Journal of Medicine Double-blind study (n=61 after run-in) comparing cyclandelate vs. pizotifen for migraine prophylaxis over 12 weeks; comparable efficacy and tolerance
8902255 1996 RCT Cephalalgia Multicentre double-blind study (n=214) vs. placebo and propranolol; cyclandelate’s calcium-antagonist profile supports prophylactic potential
9584874 1998 RCT Functional Neurology Double-blind placebo-controlled study using contingent negative variation (CNV) to probe central mechanisms of cyclandelate in migraine
11298666 2001 RCT Cephalalgia Largest trial (n=251), multicentre placebo-controlled; primary endpoint (reduction in migraine days) not met — negative result
7649498 1995 RCT Functional Neurology Double-blind study vs. propranolol and placebo; responder/non-responder analysis of cyclandelate’s prophylactic effect
3304950 1987 RCT Drugs Double-blind trial (n=40) comparing cyclandelate 800mg BID vs. flunarizine 5mg; both significantly reduced migraine symptoms vs. baseline
9829155 1998 Review Drugs Practical guide to migraine management and prevention, contextualizing prophylactic agents including calcium antagonists
10463349 1999 Review Journal of Neurology Overview of antimigraine drug treatment options for acute attacks and prophylaxis
9139407 1997 Review Therapeutische Umschau Migraine diagnosis, differential diagnosis and therapy overview

India Market Information

Cyclandelate is not currently marketed in India — 0 registered licenses on file. No dosage form, brand name, or approved indication text is available for this market.


Safety Considerations

Please refer to the package insert for safety information.

No structured key warnings, contraindications, or drug-drug interaction (DDI) data were retrievable for cyclandelate in this evidence pack — the TFDA-equivalent label lookup returned no results, and the local DDI database file was unavailable at query time. This is flagged as a Blocking data gap in the evidence pack (DG001): safety data must be obtained before this candidate can pass the initial safety screening stage.


Other Predicted Indications (Screened, Lower Priority)

For completeness, the remaining 9 TxGNN-predicted indications for cyclandelate in this evidence pack, ranked by model score:

Rank Disease TxGNN Score Evidence Level Recommendation
1 Prinzmetal angina 99.80% L5 Hold (no clinical/literature evidence)
2 Raynaud disease 99.73% L4 Research Question (1 old case-series report only)
4 Migraine with brainstem aura 99.62% L4 Research Question (indirect qEEG studies, not subtype-specific)
5 Pulmonary hypertension 99.50% L5 Hold (no evidence)
6 Gout 99.43% L5 Hold (no mechanistic rationale, likely graph noise)
7 Kyphoscoliotic heart disease 99.36% L5 Hold (no evidence)
8 Benign prostatic hyperplasia 99.08% L5 Hold (cyclandelate is not an alpha-blocker; no evidence)
9 Exostosis 99.06% L5 Hold (no mechanistic plausibility, likely graph noise)
10 Peripheral vascular disease 99.04% L3 Hold (this drug’s original historical indication, but pivotal 1984 RCT was negative)

Notably, rank 10 (peripheral vascular disease) is cyclandelate’s original indication as Cyclospasmol — and the key RCT (PMID 6364892) found no effect, which is a useful sanity check on how much weight to give TxGNN scores alone without evidence triangulation.


Conclusion and Next Steps

Decision: Proceed with Guardrails (for the migraine disorder indication)

Rationale: Migraine prophylaxis is the only indication in this candidate’s prediction set with L2 evidence (multiple historical RCTs), reflecting a genuine, previously-established off-label/international use rather than a purely computational artifact. However, the largest and most recent trial (2001) was negative, and the drug is not currently marketed in India, so this is not a “Go” without further work.

To proceed, the following is needed:

  • Obtain formal safety/label data (DG001, Blocking) — key warnings, contraindications, and DDI profile currently have no source
  • Formal DrugBank/mechanism-of-action confirmation (DG002, High) to properly ground the mechanistic rationale
  • A structured comparison of the positive (1987–1998) vs. negative (2001) trial results to assess whether efficacy is dose-, population-, or subtype-dependent
  • Confirmation of import/registration pathway feasibility, since cyclandelate has zero current registrations in India
  • The 9 lower-priority predictions (Prinzmetal angina, Raynaud disease, etc.) should remain on Hold pending any new clinical trial or literature signal — no further action needed unless new evidence emerges

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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