Cyanocobalamin

Evidence Level: L4 Predicted Indications: 1

Table of Contents

  1. Cyanocobalamin
  2. Cyanocobalamin: From Vitamin B12 Deficiency to Biotin Metabolic Disease
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Taiwan Market Information
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## Pharmacist Assessment Report

Cyanocobalamin: From Vitamin B12 Deficiency to Biotin Metabolic Disease

One-Sentence Summary

Cyanocobalamin (vitamin B12, DB00115) is a cobalamin-class vitamin whose established use is treating vitamin B12 deficiency; detailed Taiwan-approved indication text and mechanism-of-action data are not currently available. The TxGNN model predicts a possible link to Biotin Metabolic Disease with a 99.60% prediction score, but the identified evidence base (15 clinical trials, 20 publications) is largely generic B-vitamin/nutrition research rather than trials or literature specific to cyanocobalamin in biotin metabolic disease.


Quick Overview

Item Content
Original Indication Vitamin B12 deficiency (general pharmacology; Taiwan-specific approved indication text unavailable — drug not currently marketed in Taiwan)
Predicted New Indication Biotin metabolic disease
TxGNN Prediction Score 99.60% (rank 7182)
Evidence Level L4
Taiwan (TFDA) Market Status Not marketed (Not marketed)
Number of Registrations 0
Recommended Decision Hold

Why is This Prediction Reasonable?

Currently, detailed mechanism-of-action data for cyanocobalamin is not available (DrugBank query returned no structured MOA). Based on known pharmacology, cyanocobalamin is a synthetic form of vitamin B12 (cobalamin), acting as a cofactor for methionine synthase and methylmalonyl-CoA mutase; its established clinical role is correcting vitamin B12 deficiency and related megaloblastic anemia/neurological disease.

“Biotin metabolic disease” refers to disorders of vitamin B7 (biotin) metabolism, such as biotinidase deficiency and multiple carboxylase deficiency — a distinct cofactor system from cobalamin, with no established shared biochemical pathway. The TxGNN score of 99.60% is therefore more likely explained by ontology adjacency than by a direct mechanistic link: cobalamin-, folate-, biotin-, and thiamine-responsive inborn errors of metabolism are frequently grouped together in the literature and knowledge-graph structure as a single “vitamin-responsive/cofactor-responsive IEM” cluster, which can inflate similarity scores between individually unrelated cofactor systems.

Given this, the prediction should be treated as a hypothesis requiring manual verification of the TxGNN disease-node definition, not as evidence of a genuine pharmacological relationship. This assessment is reinforced by the absence of any clinical trial or publication directly testing cyanocobalamin in biotin metabolic disease patients.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT05687474 N/A Completed 6,824 Universal newborn genomic screening panel (126 treatable genetic diseases); may include biotinidase deficiency but is a screening, not treatment, study — not specific to cyanocobalamin (Grade C).
NCT01474486 N/A Completed 40 Multi-micronutrient palliative intervention in CHF patients; not specific to biotin metabolic disease or cyanocobalamin alone (Grade C).
NCT04312152 N/A Unknown 200 Cross-over RCT of Q10 ubiquinol + multivitamin B/E in autism (incl. Phelan-McDermid syndrome); no clear link to biotin metabolic disease (Grade C).
NCT03444155 N/A Completed 30 Pilot comparison of natural vs. synthetic vitamin B-complex bioavailability; not disease-specific (Grade C).
NCT01173315 Phase 2 Completed 75 Vitamin/mineral supplementation for neuropathy/nephropathy in type 2 diabetes; unrelated to biotin metabolic disease (Grade C).
NCT04586348 Phase 4 Active, not recruiting 794 Prenatal iodine supplementation and neurodevelopment; no direct relevance to cyanocobalamin or biotin metabolic disease (Grade C).
NCT03360435 N/A Completed 99 Transdermal vitamin absorption in post-bariatric-surgery patients; general micronutrient study, not disease-specific (Grade C).
NCT02426775 Phase 3 Completed 33 Long-term effectiveness of carglumic acid in propionic/methylmalonic acidemia; related organic acidemias but no direct cyanocobalamin-biotin link (Grade C).
NCT00572741 N/A Completed 39 Targeted nutritional intervention for oxidative stress/methylation defects in autism; not biotin-metabolic-disease specific (Grade C).
NCT01558193 N/A Completed 202 Multivitamin/mineral ± fatty acid supplementation and behavior; unrelated to target indication (Grade C).

(An additional 5 trials returned by the search were not yet relevance-graded — NCT02302729, NCT03655223, NCT05832190, NCT04067921, NCT01643187.)


Literature Evidence

PMID Year Type Journal Key Findings
23622402 2013 Review (Tier 1) Handbook of Clinical Neurology Reviews vitamin-responsive disorders including cobalamin, folate, biotin, B1, and E — the most directly relevant source, discussing cobalamin and biotin as parallel but distinct cofactor-dependent disease categories.
958746 1976 Review/Case Series (Tier 2) Pediatric Clinics of North America Discusses megavitamin-responsive aminoacidopathies where B-vitamin cofactors (including B12 and biotin) activate deficient apoenzymes.
7027768 1981 Review (Tier 2) Acta Vitaminologica et Enzymologica Reviews vitamins implicated in inborn metabolic errors via malabsorption, metabolic errors, or vitamin-dependent syndromes.
11031989 2000 Review (Tier 2) Ryoikibetsu Shokogun Shirizu Japanese-language review of vitamin dependency syndromes; abstract not available.
38203763 2024 Review (Tier 2) International Journal of Molecular Sciences Reviews vitamin B12’s cofactor role (methylmalonyl-CoA/biotin-adjacent pathway, methionine synthesis) and neurological effects of deficiency.
25388747 2015 Review (Tier 3) Endocrine, Metabolic & Immune Disorders Drug Targets Reviews vitamins (including B-group and biotin) in type 2 diabetes; general, not disease-specific.
29173522 2017 Review (Tier 3) Gastroenterology Clinics of North America Reviews vitamin/mineral deficiencies in IBD; general micronutrient context.
7015958 1980 Review (Tier 3) Annals of the New York Academy of Sciences Reviews interactions among B-complex vitamins including thiamin and riboflavin.
36476407 2023 Preclinical (Tier 3) The Journal of Endocrinology Rat study: B12 deficiency induces glucose intolerance and a prediabetic-like phenotype.
1368195 1992 Review — industrial process (Tier 3) Journal of Chemical Technology and Biotechnology Reviews biotechnological production of vitamins/coenzymes; not clinically relevant.

(10 additional publications were returned but remain unclassified/pending review.)


Taiwan Market Information

No marketed cyanocobalamin products are currently registered with TFDA under this evidence pack (0 licenses, market status: Not marketed / Not marketed).


Safety Considerations

Drug Interactions: 313 total documented interactions on file. Representative interactions include:

  • Minor — reduced absorption with acid-reducing agents: Famotidine, Ranitidine, Ranitidine (bismuth citrate), Cimetidine, Nizatidine (H2 blockers); Omeprazole, Esomeprazole, Pantoprazole, Lansoprazole, Rabeprazole, Dexlansoprazole (PPIs); and Potassium chloride.
  • Unknown clinical significance — Metformin, Glimepiride, Rosiglitazone, Acarbose, Doxycycline, Morphine, Mesalazine, Sucralfate.

Taiwan-specific label warnings and contraindications are not currently available in this evidence pack; please refer to the package insert once TFDA labeling data is obtained.


Conclusion and Next Steps

Decision: Hold

Rationale: The TxGNN score is high, but the review indicates it is likely driven by ontology adjacency (cofactor-responsive IEMs being clustered together) rather than a genuine biochemical link between cobalamin and biotin pathways. No clinical trial or publication was found that directly tests cyanocobalamin in biotin metabolic disease — all trials are Grade C (tangential) or unassessed, and the strongest literature source (PMID 23622402) discusses cobalamin and biotin as parallel, not overlapping, disorders.

To proceed, the following is needed:

  • Cyanocobalamin MOA data from DrugBank (DG002, High severity)
  • TFDA label warnings/contraindications (DG001, Blocking — required before any S1 safety screening)
  • Manual review of the TxGNN “biotin metabolic disease” node definition to rule out an ontology-clustering artifact
  • Targeted search for any case reports or trials specifically evaluating cobalamin/B12 supplementation in biotinidase deficiency or multiple carboxylase deficiency patients

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



Copyright © 2026 藥提醒科技有限公司 (yao.care). For research purposes only. Not medical advice.

This site uses Just the Docs, a documentation theme for Jekyll.